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4. Raisz, L. G., Sandberg, A. L., Goodson, J. M., Simmons, H. A. and Mergenhagen, S. E., Complement-dependent stimulation of prostaglandin synthesis and bone resorption. Science, 1974, 185, 789791. Dowsett, M., Eastman, A. R., Easty, D. M., Easty, G. C., Powles, T. J. and Neville, A. M., Prostaglandin mediation of collagenase induced bone resorption. Nature, 1976, 263, 7274. Goldhaber, P., Rabadjija, L., Beyer, W. R. and Kornhauser, A., Bone resorption in tissue culture and its relevance to periodontal disease. J. Am. Dent. Assoc., 1973, 87, 10271033. Harris, M., Jenkins, M. V., Bennet, A. and Wills, M. R., Prostaglandin production and bone resorption by dental cysts. Nature, 1973, 245, 213215. Goodson, J. M., McClatchy, K. and Revell, C., Prostaglandininduced resorption of adult rat calvarium. J. Dent. Res., 1974, 53, 670677. Yamasaki, K, Miura, F. and Suda, T., Prostaglandin as a mediator of bone resorption induced by experimental tooth movement in rats. J. Dent. Res., 1980, 59, 16351642. Davidovitch, Z., Nicolay, O. F., Ngan, P. W. and Shanfeld, J. L., Neurotransmitters, cytokines, and the control of alveolar bone remodeling in orthodontics. Dent. Clin. North Am., 1988, 32, 411435. Chumbley, A. B. and Tuncay, O. C., The effects of Indomethacin an aspirin-like drug ; on the rate of tooth movement in cats. Am. J. Orthod., 1986, 89, 312314. Yamasaki, K., Shibata, Y. and Fukuhara, T., The effect of prostaglandins on experimental tooth movement in monkeys Macaca fuscata ; . J. Dent. Res., 1982, 61, 14441446. Yamasaki, K., Shibuta, Y., Imai, S., Tani, Y., Shibasaki, Y. and Fukuhara, T., Clinical application of prostaglandin E1 PGE1 ; upon orthodontic tooth movement. Am. J. Orthod., 1984, 85, 508 Lee, W., Effect of prostaglandins on tooth movement. Am. J. Orthod. Dentofac. Orthop., 1990, 98, 231241. Leiker, B. J., Nanda, R. S., Currier, G. F., Howes, R. I. and Sinha, P. K., The effects of exogenous prostaglandins on orthodontic tooth movement in rats. Am. J. Orthod. Dentofac. Orthop., 1995, 108, 380388. Bhalajhi, S. I. and Shetty, V. S., The effect of prostaglandin E2 on tooth movement in young rabbits. J. Indian Orthod. Soc., 1996, 27, 8592. Kehoe, M. J., Cohen, S. M., Zarrinnia, K. and Cowan, A., The effect of acetaminophen, ibuprofen and misoprostol on prostaglandin E2 synthesis and the degree and rate of orthodontic tooth movement. Angle Orthod., 1996, 66, 339350. Sekhavat, A. R., Mousavizadeh, K., Pakshir, H. R. and Aslani, F. S., Effect of misoprostol, a prostaglandin E1 analog, on orthodontic tooth movement in rats. Am. J. Orthod. Dentofac. Orthop., 2002, 122, 542547. Seifi, M., Eslami, B. and Saffar, A. S., The effect of prostaglandin E2 and calcium gluconate on orthodontic tooth movement and root resorption in rats. Eur. J. Orthod., 2003, 25, 199204. Collins, M. K. and Sinclair, P. M., The local use of vitamin D to increase the rate of orthodontic tooth movement. Am. J. Orthod. Dentofac. Orthop., 1988, 94, 278284. Kale, S., Kocadereli, I., Atilla, P. and Asan, E., Comparison of the effects of 1, 25-dihydroxycholecalciferol and prostaglandin E2 on orthodontic tooth movement. Am. J. Orthod. Dentofac. Orthop., 2004, 125, 607614. The CyFlow Counter is a fully-equipped portable parallel signal processing for each of the optical desktop flow cytometer with laser excitation in channels with selectable linear, 3- or 4-decade green. It analyses up to three optical parameters logarithmic scale pulse height, area and width analysis for SSC and 2 fluorescences channels ; plus time parameter. It performs both fluorescence analysis doublet discrimination 16 bit analog-to-digital converters, trigger on and true volumetric absolute cell counting without the need for reference beads. any parameter or parameter combinations.

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The Revolving Drug Fund Manual A revolving drug fund RDF ; starts with a sum of money contributed by the government, donors, the community, or from within the organization. The funds are used to purchase an initial stock of essential and commonly used drugs to be sold, ideally at prices sufficient to replace the stock of drugs and ensure a continuous supply. The ultimate objective of establishing an RDF is to create a self-sustaining source of funding to ensure access to a continuous supply of quality drugs at an affordable price. 156. Ryan RE Sr, Ryan RE Jr. Migraine prophylaxis: a new approach. Laryngoscope. 1981; 91 9 pt 1 ; 1501-1506. 157. Masel BE, Chesson AL, Peters BH, Levin HS, Alperin JB. Platelet antagonists in migraine prophylaxis: a clinical trial using aspirin and dipyridamole. Headache. 1980; 20 1 ; : 13-18. 158. Couch JR, Bearss CM, Verhulst S. Fenoprofen in migraine prophylaxis. Headache. 1987; 27 5 ; : 289. 159. Diamond S, Solomon GD, Freitag FG, Mehta ND. Fenoprofen in the prophylaxis of migraine: a double-blind, placebo-controlled study. Headache. 1987; 27 5 ; : 246-249. 160. Mikkelsen BM, Falk JV. Prophylactic treatment of migraine with tolfenamic acid: a comparative double-blind crossover study between tolfenamic acid and placebo. Acta Neurol Scand. 1982; 66 1 ; : 105-111. 161. Solomon GD, Kunkel RS. Flurbiprofen in the prophylaxis of migraine. Cleve Clin J Med. 1993; 60 1 ; : 43-48. 162. Carrieri PB, Orefice G, Sorge F. A double-blind placebo-controlled trial of indobufen in the prophylaxis of migraine. Acta Neurol Scand. 1988; 77 6 ; : 433-436. 163. Anthony M, Lance JW. Indomethacin in migraine. Med J Aust. 1968; 1 2 ; : 56-57.
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17. SALBUTAMOL INFUSION - OBSTETRIC CONT ; Precautions and Side Effects cont ; 2. Side effects include palpitations, tachycardia, tremor, hypotension, pulmonary oedema, cardiac arrhythmias, myocardial ischaemia, hypokalaemia, hyperglycaemia, flushing, headache, dizziness, anxiety, nausea and vomiting. 3. Use with caution in patients with heart disease, diabetes or hypertension 4. Absolute obstetric contraindications include fetal death in utero and severe antepartum haemorrhage. 5. May be use when relative contraindications exist, if risks outweighed by the risk of delivery during transport. Special Notes: 1. In this RFDS protocol both the rate of increase of infusion and the maximum limits are greater when compared with the King Edward Memorial Hospital protocol. This reflects the different risks involved in the delivery of the pre-term infant outside the tertiary Hospital setting. Care must be exercised at high doses. 2. Alternatives adjuncts to Salbutamol infusion for tocolysis include Nifedipine, Ritodrine, Indomethacin and GTN. Consult Clinical Guideline on Preterm Labour. Fig. 4. Dose-response to acute hypoxia in isolated lungs. a ; Rats investigated at the age of 10 weeks Groups I-10 n 5 ; and C-10 n 5 ; , see table 1 ; . b ; Fourteen week old rats recovering from hypoxic exposure Groups C-14 n 6 ; and I-14 n 5 ; , see table 1 ; . Vertical bars represent SEM. Two-way analysis of variance ANOVA ; indicated a significant relation between the pressor response and degree of acute hypoxia in all groups. The responses P ; of Groups C-14 and I-14 are significantly bigger than those of Groups C-10 and I-10, respectively, but there is no significant difference between the control and indomethacin-treated groups. : Group C-10; q : Group I-10. - - : Group C-14; v : Group I-14 and imdur.
SPORANOX * Misc. Anti-Infectives # clindamycin dapsone ees sulfisoxazole metronidazole nitrofurantoin sulfisoxazole GANTRIS PED ; tmp smx OPHTHALMIC AGENTS Ophthalmic Anti-Infective Agents bacitracin-polymyxin b bacitracin ciprofloxacin hcl erythromycin gentamicin sulfate polymyxin b-trimethoprim sulfacetamide sodium tobramycin sulfate trifluridine VIRA-A Ophthalmic Antiallergic cromolyn sodium $ naphazoline w pheniramine ; $ phenylephrine $ VASOCON-A Ophthalmic Beta Blockers levobunolol hcl metipranolol timolol maleate Ophthalmic Prostaglandins XALATAN Ophthalmic Selective Alpha Adrenergic Agonists brimonidine tartrate 0.2% PAIN MANAGEMENT Non-Narcotic Agents $ acetaminophen ASA-APAP-caffeine $ aspirin butalbital-APAP-caffeine butalbital-aspirin-caffeine choline & mag salicylate salsalate # tramadol NSAIDs diclofenac sodium etodolac fenoprofen $ ibuprofen indomethacin nabumetone naproxen piroxicam 20 mg sulindac COX-2 Inhibitors.

John Kraft; 1 Scott Walsh; 2 1 Faculty of Medicine, University of Toronto, Toronto, ON, Canada 2 Division of Dermatology, University of Toronto, Sunnybrook and Women's College Health Sciences Centre, Toronto, ON, Canada Introduction: Follicular mucinosis FM ; is an uncommon chronic inflammatory disease of unknown aetiology with controversy surrounding its nomenclature and association with mycosis fungoides MF ; . It characterized by mucin deposits around hair follicles and sebaceous glands. Primary variants are benign and include Pinkus' and urticaria-like FM. Secondary FM can be associated with malignant lymphoproliferative disorders i.e. MF, Hodgkin's disease, acute and chronic lymphocytic leukemia ; but also with many benign conditions i.e. insect bites, lupus erythematosus, spongiotic dermatitis, Goodpasture's syndrome ; . Treatment of FM is challenging. Limited anecdotal evidence exists to support the use of topical and systemic corticosteroids, isotretinoin, indomethacin, dapsone, minocycline, and phototherapy and sorbitrate. Other than exercise, non-drug treatment include: cognitive-behavioral treatment, biofeedback, hypnotherapy relaxation techniques and multi-disciplinary pain management programs. Most studies involving these treatment modalities have been positive. Diclofenac, an acetic acid derivative, is a popular alternative as a first or second-line agent; indomethacin, which is felt to have a superior anti-inflammatory action, is associated with a higher incidence of side effects and is often reserved as a third-line agent and imipramine.

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Hyoscyamine . 9 I ibuprofen . 1, 3 IMITREX. 3 indomethacin . 1, 3 INFERGEN . 11 INTAL . 13 INVEGA . 5 INVIRASE. 5 ipratropium bromide . 13 isoniazid . 4 isosorbide dinitrate . 7 isosorbide mononitrate . 7 itraconazole . 3 J JANUMET. 6 JANUVIA . 6 K KEPPRA . 2 KETEK . 1 ketoconazole . 3 ketoconazole cream . 3 ketoconazole shampoo . 3 ketotifen fumarate opthalmic . 12 L lactulose . 9 LAMICTAL. 2 LAMISIL . 3 leucovorin calcium . 4 LEUKERAN . 4 leuprolide acetate . 10 LEVAQUIN. 1 levobunolol . 12 levothyroxine . 10 levoxyl . 10 lidocaine. 8 lidocaine viscous. 1 LIDODERM . 1 LIPITOR . 7 lisinopril . 7 lithium . 5, 6 LORABID . 1 LOTRONEX . 9 lovastatin . 7 LOVENOX . 6 LUMIGAN . 12 LYSODREN . 10 and tofranil. Shortly, allowing for any rebound acidity to be detected within 5 h of indomethacin administration. Effect on serum gastrin level In another set of experiments, animals were divided into six groups, each of 6 rats, to determine the serum gastrin level. Animals were fasted for 18 h before subjecting them to experimentation. One group of rats was kept as control, and the others were injected intraperitoneally with 10 mg kg indomethacin. One hour later, the rats were given the drugs mentioned above in the pre-selected doses. Blood was collected from the retro-orbital plexus of each animal using fine heparinized capillary tubes, 2 and 5 h after indomethacin administration. The collected blood was centrifuged and the serum was separated and kept frozen until assayed for gastrin using a Gamma Dab Gastrin 125I radioimmunoassay kit DiaSorin Stillwater, Minnesota, USA ; . Statistical analysis Statistical analysis was carried out using one way analysis of variance ANOVA ; followed by the least significant difference LSD ; test. Values are given as means7S.D. and po0: 05 was considered to be statistically significant.
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Examine the endogenous vasoactive properties during CBDLinduced cirrhosis in rats. Our primary observations are that CBDL kidneys have 1 ; reduced vasoconstriction in response to phenylephrine in the presence of NOS, cyclooxygenase, and CYP 450 inhibition; 2 ; exaggerated vasodilation to ACh; and 3 ; increased production of vasodilating arachidonic acid, 11, 12-EET and 14, 15-EET. Exaggerated AChinduced renal vasodilation in vivo and in isolated, perfused kidneys from cirrhotic rats has been reported once before 16 ; . As observed in the present study, this exaggerated renal vasodilation cannot be fully accounted for by NO, as demonstrated using L-NAME to inhibit NOS activity. We used several physiologic and biochemical approaches to identify the NO-independent source of renal vasodilation. Prostaglandins are known to cause NO-independent renal vasodilation 26 ; . To examine their role in the NO-independent component of vasodilation, we used indomethacin together with L-NAME to inhibit prostaglandin and NO production, respectively. While this treatment largely reduced the exaggerated ACh-dependent vasodilation in the CBDL kidneys, a and isoflavone and indomethacin.
We analyzed data in 138, 714 patients with NSTE ACS treated We analyzed data in 138, 714 patients with NSTE ACS treated at 547 hospitals participating in the CRUSADE Can Rapid Risk at 547 hospitals participating in the CRUSADE Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Stratification of Unstable Angina Patients Suppress Adverse Outcomes with Early Implementation of ACC AHA Guidelines ; Outcomes with Early Implementation of ACC AHA Guidelines ; Quality Initiative from January 2002 through December 2005. All Quality Initiative from January 2002 through December 2005. All patients had ischemic ECG alterations and or positive cardiac patients had ischemic ECG alterations and or positive cardiac injury markers. injury markers. Patients were categorized into 4 treatment strategies: Patients were categorized into 4 treatment strategies: --Medical therapy only Med Only ; Medical therapy only Med Only ; --Cardiac catheterization, no revascularization Cardiac Cath ; Cardiac catheterization, no revascularization Cardiac Cath ; --Percutaneous coronary intervention PCI ; Percutaneous coronary intervention PCI ; --Coronary artery bypass graft surgery CABG ; Coronary artery bypass graft surgery CABG ; Trends in the application of the 4 treatment strategies during the Trends in the application of the 4 treatment strategies during the study period 2002-2005 ; were examined in relation to the study period 2002-2005 ; were examined in relation to the following features of each group: following features of each group: --Clinical characteristics Clinical characteristics --Use of medical therapies Use of medical therapies --Clinical outcomes Clinical outcomes.
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162 USING THE WHO ANATOMICAL THERAPEUTIC CHEMICAL DEFINE DAILY DOES ATC DDD ; DRUG CLASSIFICATION SYSTEM ATC DDD ; TO COMPARE NON-STEROIDAL ANTI-ANFLAMMATORY DRUGS AVAILABLE IN ATLANTIC CANADA N Ninos, S Bowles, I Sketris Institutions: Dalhousie University, Halifax, NS Funding Source: Canadian Institutes of Health Research Undergraduate Pharmacy Studentship BACKGROUND: Many Non-Steroidal Anti-Inflammatory Drugs NSAIDs ; are on the Canadian market, each with different characteristics, indications and prices. We compared NSAIDs by ATC class, indication and price DDD. METHODS: NSAIDs on the Canadian market were identified from the Health Canada Drug Product Database. Indications for each chemical entity were obtained from product monographs in the 2002 CPS. The 2002 Atlantic Pharmaceutical Services Incorporated Pricing Guide was used to obtain prices. Drugs were grouped based on the 2002 WHO ATC DDD classification system and the price DDD was determined. Price DDD was classified as low price median ##TEXT##.50 medium price median ##TEXT##.50 high price median ; . RESULTS: Twenty-one chemical entities were identified, representing 350 individual NSAID products DINs ; . Indications for therapy varied with ATC groups B, C, E, X and H indicated for rheumatoid arthritis and osteoarthritis with other specific groups indicated for conditions such as dysmenorrhea and dental surgery. Fourteen NSAIDs were multisource products. Piroxicam and diclofenac were available from the greatest number of manufacturers 12 and 10 respectively ; . Using the metric price DDD, the least expensive NSAIDs for arthritic, musculoskeletal conditions and pain were ibuprofen, naproxen and indomethacin. CONCLUSION: NSAIDs use by drug class, indication, price DDD and manufacturer were compared. When used in conjunction with appropriate clinical decision-making, these results can assist clinicians and policy makers in selecting NSAIDs. KEY WORDS: Defined daily dose; anatomical therapeutic chemical classification; non-steroidal anti-inflammatory drugs; pricing.
MATERNAL DRUG THERAPIES FOR SPECIFIC CONDITIONS OF PREGNANCY, DELIVERY AND POSTPARTUM R003 ; DRUGS FOR CONDITIONS PREG PP ; For Use With: Delivered Undelivered Postpartum Found on the `PRENATAL RECORD'. Choose as many as are indicated; 100 200 300 Adalat nifedipine ; for premature labour ASA Therapy Low dose aspirin therapy for Lupus and or any other autoimmune conditions ; Atosiban for premature labour Hemabate for Postpartum Hemorrhage Indocid Indomethacin ; for premature labour Indocid Indomethacin ; for tx of Polyhydramnios Magnesium sulfate therapy MgSO4 ; for hypertension or seizures, e.g. Eclampsia prophylaxis or treatment ; . Magnesium Sulfate MgSO 4 ; for premature labour Pentaspan for Postpartum Hemorrhage Terbutaline Bricanyl ; for premature labour Ventolin for premature labour Other Drugs for Specific Pregnancy, Delivery or Postpartum conditions.
Jach DT, Piper JM, Glebatis DM. Oral contraceptives and congenital limb reduction defects. N Engl J Med 1974; 291: 697-700 Jackson AE, Curtis P, Amso N, Shaw RW. Exposure to LHRH agonist in early pregnancy following the commencement of mind-luteal buserelin for IVF stimulation. Human Reprod 1992; 7: 1222-1223. Jackson D, Cockburn A, Cooper Dl et al. Clinical pharmacology and safety evaluation of timentin. J Med 1985; 79: 44-55. Jackson GL. Treatment of hyperthyroidism in pregnancy. Pa Med 1973; 76: 56-57. Jackson N, Shukri A, Ali K. Hydroxyurea treatment for chronic myeloid leukaemia during pregnancy. Br J Haematol 1993; 85: 203-204. Jacobs AJ, Marchevsky A, Gordon RE, et al. Oat cell carcinoma of the uterine cervix in a pregnant woman treated with cis-iamminedichloro -platinum. Gynecol Oncol 1980; 9: 405-410. Jacobs C, Donaldson SS, Rosenberg SA, Kaplan HS. Management of the pregnant patient wiyh Hodgkin's disease. Ann Intern Med 1981; 95: 669-675. Jacobs D. Imipramine tofranil ; . S Afr Med J 1972; 46: 1023. Jacobs D. Maternal drug ingestion and congenital malformations. S Afr Med J 1975; 49: 2073-2080. Jacobson BD. Hazards of norethindrone therapy during pregnancy. J Obstet Gynecol 1962; 84: 962-968. Jacobson JF. A randomized controlled trial comparing amoxicillin and azithromycin for the treatment of Chlamydia trachomatis inpregnancy. J Obstet Gynecol 2001; 184: 1352-1354. Jacobson JF. A randomized controlled trial comparing amoxicillin and azithromycin for the treatment of Chlamydia trachomatis inpregnancy. J Obstet Gynecol 2001; 184: 1352-1354. Jacobson JM, Hankins GV, Young RL, Hauth JC. Changes in thyroid function and serum iodine levels after prepartum use of a povidone-iodine vaginal lubricant. J Reprod Med 1984; 29: 98-100. Jacobson SJ, Jones KL, Johnson K et al. Prospective multicentre study of pregnancy outcome after lithium exposure during first trimester. Lancet 1992; 1: 339: Jacqz-Aigrain E, Guillonneau M, Boissinot C, et al. Maternal and neonatal effects of indomethacin administrated during pregnancy. Apropos of 18 cases. Arch Fr Pediatr 1993; 50: 307-312 Jaeggi E, Fouron JC, Fournier A, et al. Ventriculo-atrial time interval measured on M mode echocardiography: A determining element in diagnosis, treatment, and prognosis of fetal supraventricular tachycardia. Heart 1998; 79: 582-587. Jaffe P, Liberaman MM, McFadyen I, Valman HB. Incidence of congenital limb-reduction deformities. Lancet 1975; 1: 526-527. Jager-Roman E, Deichl A, Jakob S, et al. Fetal growth, major malformations, and minor anomalies in infants born to women receiving valproic acid. J Pediatr 1986; 108: 9971004. Jahn A, Blode H, Schutzel H, Gunzel P. Developmental toxicology data of cyproterone acetate their relevance for clinical safety assessment. Teratology 1996; 53: 31A. Jahn AF, Ganti K. Major auricular malformations due to Accutane isotretinoin ; . Laryngoscope 1987; 97: 832-835. Jain A, Venkataramanan R, Fung J et al. Pregnancy after liver transplantation under tracrolimus. Transplantation 1997; 64: 559-565. Jain AB, Reyes J, Marcos A, et al. Pregnancy after liver transplantation with tacrolimus immunosuppression: a single center's experience update at 13 years. Transplantation 2003; 76: 827-832. Jaiswal S, Coombs RC, Isbister GK. Paroxetine with drawal in a neonate with historicl and laboratory confirmation. Eur J Pediatr 2003; 162: 723-724. James V, Chiccarelli F, Dougherty W, et al. Preclinical toxicology studies on mitoxantrone and bisantrene. In Rozencwieg M, Van Hoff DD, Staquet MJ. New Anticancer Drugs: Mitoxantrone and Bisantrene. New York Raven Press 1983. James WH. Anencephaly and ovulation stimulation and subfertility. Lancet 1974; i: 1353.
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