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In contrast to testing for urinary incontinence, "dynamic" testing for faecal incontinence is not yet well defined. The causes of faecal incontinence fall into two broad interactive categories: factors that are associated with the nature of stool and the frequency of bowel movements; and anatomical or functional disturbances of the anorectal area. The stool features commonly associated with incontinence are loose and frequent bowel movements, that is, diarrhoea. However severe constipation and packing of the rectum with stool can lead to "overflow incontinence". The local anorectal mechanisms that maintain continence include the specialized anatomy and innervation of the region, the sensory-motor function of the rectum, and the resting, voluntary and reflex function of the musculature both smooth and striated ; of the anal sphincter apparatus. [431] Other factors such as cognitive ability, mobility, and drugs can impact the anorectal continence mechanisms and or the patient's ability to defaecate normally. Testing of the anorectal continence mechanisms has included many approaches directed at different anatomical and physiological sensory-motor aspects, and functional behaviours. [431-433] Although urodynamics can be considered the study of lower ; urinary tract function and dysfunction by any relevant method, it has developed primarily into assessment of pressure, volume and flow, electromyogram activity and responses of the urethral sphincter, and bladder sensation. These measurements are made in relation to dynamic activities such as bladder filling, micturition, and contraction of the urethral sphincter and pelvic floor musculature. It is reasonable at this stage to approach dynamic testing for faecal incontinence in the same way. That is, to assess those tests that measure pressure, volume and or flow, electromyogram activity and responses of the external anal sphincter, and rectal sensation these in relation to dynamic activities such as rectal distension and contraction, defaecation, and contraction of the anal sphincter and pelvic floor musculature. Perhaps a term such as "faecodynamics" will arise.

This letter mitten in behalf of Dr. Raquel Arancta who ie applying for a Staff position in Pediatric Pulmonology at the U. Manitoba Children's Hospital at Winnipeg. As you very will know she had her 2 year Fcllowship in Pediatric Pulmonolgy at your hospital in the p a t Upon amval to Manila, Iappointed her as assistant head of our section in Pulmonology at the Makita Medical Center and she hiid unselfishly shared her knowledge gleaned fiom her training and expertise while with you for 2 years. Tg mice are transgenic mice harboring multiple copies approximately 20 ; of a mutant mouse v-Ha-ras gene under control of a -globin promoter Leder et al. 1990 ; . The transgene is silent, until activated by wounding, UV irradiation or specific chemical exposure. The model can be used testing chemical compounds on the skin after topical application. In response to these treatments, Tg mice develop squamous cell papillomas or carcinomas. Also oral administration results in tumor responses in the skin of these mice, in addition, tumors in the forestomach are observed Tennant et al. 2001 ; . For testing however, the dermal application is currently the most acceptable route. Interestingly, the Tg model responds to topical application with tumor promoters such as phorbol esters. Therefore, the Tg ras model can be viewed as a genetically initiated model, and as such, is expected to respond to both genotoxic and non-genotoxic agents. Spontaneously, less than 4% of the control animals develop tumors at the site of application when treated with the solvent, other tumors occur in a broader range of tissues with low incidence Tennant et al. 2001 ; . Major drawback of this model however is that the mechanism of response by the Tg model to chemical carcinogens is not well understood. Robert E. Adamiak, SSN: 383-38-9974 vs. DaimlerChrysler Corporation training, that pays a wage equivalent to his maximum income. Once those two conditions are met on the record, disability is established, and it is then incumbent upon the Defendant to refute such assertion with evidence of its own. In this case both Plaintiff and both medical experts testified credibly that Plaintiff has severe restrictions in his bodily movements such that the only work Plaintiff may be able to perform is sedentary. Plaintiff also testified credibly that at the time he ceased working for Defendant DC in 1990, there was no work to be had within his physical limitations. Under Stokes, supra, I understand that this is sufficient testimony upon which to rest a finding of disability Defendant having come forth with no real evidence to counter such holding ; : I so find Plaintiff disabled under 301 4 ; of the Act. The next question to answer involves causation; that is, how did Plaintiff develop his disability? Defense expert indicates that Plaintiff's present condition of severe degenerative disc disease of the left hip is due to developmental factors, something that occurred to Plaintiff as a pre-teen, perhaps, and has absolutely nothing to do with the one trauma at work the 6 27 1989 slip fall ; of which Dr. Drouillard took a history from Plaintiff. Plaintiff's medical expert, on the other hand, opines that Plaintiff had a series if slip fall insults to his left hip throughout his career at Defendant, which caused an avascular necrosis in the left hip, leading to its present collapsed state. The diagnosis of avascular necrosis is also supported in the records by Dr. Whitman and Dr. Roy. Under these circumstances, especially since I find Dr. Drouillard's history from Plaintiff incomplete and inaccurate and thus not a sufficient basis from which to form a medico-legal opinion ; I choose to accept Dr. Belen's explanation of causation for Plaintiff's left degenerative disease of the hip, which ties the origin of that condition directly to the series of traumas Plaintiff underwent at work, his development of avascular necrosis therefrom, and ultimately Plaintiff's disability. I hold, in short, that Plaintiff's current disability is causally related to Plaintiff's employment. Having found a work-related disability in Plaintiff, I now consider the question of total and permanent disability for reasons of industrial loss of use of both lower and lamictal. Non-steroidal anti-inflammatory drugs are sometimes used but should be avoided in the third trimester because they can have serious health effects on the fetus and newborn. Some health researchers say the trend has crossed onto college campuses and lamotrigine. Drug is of limited proven value in treating chronic pain. Of greater concern is the lack of data on the effect of decreased prescribing of opioid analgesics. In the absence of such data, it is impossible to say whether such notification helped or harmed the physicians' patients.
Back then the meds wern't out there and i believe it was thought back then ; smoking good for your health and levothyroxine. Hospital pharmacy volume 42, number 3, pp 262270 2007 wolters kluwer health, inc.
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Available dose & quan : 30 3 1mg tabs; 30 3 x 10 ; 1mg tabs; 1mg 30; 1mg tablets; 20 tablets; 50 tablets; 100 capsules; 20 capsules; 50 capsules; medication labelled produced by ketasma ketotifen, zaditen ; rx free manufactured sun pharma 1mg tabs 30 3 x ketotifen without prescription , zaditen not the prevention asthma and duration type asthma symptoms when lead may day reduction along a of to frequency, the requirements ketotifen in other it attacks and lithium. Matic relief produced by immediate histamine receptor antagonists, coupled with the long-term disease-modifying benefit of mast cell stabilizers. Olopatadine Patanol ; acts as a mast cell stabilizer, H1 receptor antagonist and inhibits cytokine secretion. Azelastine Optivar ; and Nedocromil Alocril ; have some inhibitory effect on inflammatory cells. Ketotifen Zaditor ; is a drug that acts as a histamine receptor antagonist and mast cell stabilizer. Ketotifen is a relatively selective, noncompetitive antagonist of histamine H1 receptors and a mast cell stabilizer, inhibiting the release of inflammatory mediators from mast cells. In addition, it has been shown to modulate the actions of eosinophils via several distinct mechanisms other than mast cell stabilization, including a direct effect on the endothelium, inhibiting the synthesis and expression of cellular adhesion molecules that play a key role in the recruitment of eosinophils; antagonism of the activity of platelet activating factor, thereby inhibiting the recruitment and activation of eosinophils; inhibition of eosinophil chemotaxis and activation induced by eotaxin and IL-5; and a direct stabilizing effect on eosinophils, thus preventing degranulation.6 In antigen challenge tests, both olopatadine and ketotifen were found to be very efficient in r a reducing signs and symptoms of seasonal allergic conjunctivitis within minutes, including redness, itching, tearing, chemosis, eye lid swelling and mucous discharge.7 Nonsteroidal anti-inflammatory drugs NSAIDs ; This class of drugs functions by inhibiting prostaglandin PG ; production. PGE2 and PGI 2 are extremely pruritogenic to the conjunctival mucosa. NSAIDs diminish ocular itching and hyperemia by inhibiting these factors. It may take 2 weeks of topical use to have an effect. Topical corticosteroids Corticosteroids reduce the transcription of proi n flammatory genes eg, the gene for phospholipase A2 ; and thus reduce the amount of PGs produced. However, corticosteroids also promote the release of phospholipase A2 inhibitor Lipocortin ; from leukocytes. There is no clear understanding of the mech anism at the cellular and plasma levels.8 G l u icoids inhibit the production of interleukins ILs ; , among them IL-4 and IL-5 that are the primary ILs produced by ocular surface mast cells. Steroids also reduce transcription of eotaxin and may induce apoptosis of eosinophils and T-c e l l s steroids do not have an immediate effect on ocular itching caused by allergies. Their effect is based on attenuating the late phase response. In view of their potential side effects, including cataracts, increased intraocular pressure, and corneal melts, corticosteroids are typically reserved for patients not responsive to other therapy or fo r use in the severe forms of allergy, such as AKC or VKC, and acute exacerbations of VKC. The regimen is a high dose of dexamethasone or prednisolone 8 times daily for 1 week and then rapid tapering. Topical mast cell stabilizers or a combined medication should be started concurrently.

Administered an intranasal sterile physiological saline solution placebo ; for 2 weeks in the same regimen as the consecutive vasopressin administration. The study was carried out under double blind controlled conditions. Follow-up investigation and repeated administration of arginine-vasopressin were carried out in these same 26 patients with aphasia 1-2 years after completion of the first course. For the interpretation of the effect of vasopressin, speech was examined as a functional system, the afferent, central and efferent components of which correspond to the processes of perception, comprehension, and production of speech, i.e., the sensory, integrative, and motor components ; . In order to assess these components, specific neuropsychological tests were used to rate the separate components of speech. The sensory component of speech was characterized by indices such as: speech comprehension, writing by dictation, phonetic analysis, silent reading, and its comprehension. The integrative component, reflecting the state of acousticoverbal memory, was evaluated by the retention of auditory verbal series repetition of a series of vowels and consonants, words and sentences ; , object naming, and matching picture with text. Changes in the separate components of speech were assessed by comparing the total arithmetic mean values prior to the administration of the neuropeptide argininevasopressin, after placebo and subsequent to the course of treatment after 1 -2 months. During the period of investigation, the patients continued to take medication previously prescribed, such as antiaggregant and or hypotensive drugs. To identify speech components that changed significantly under the influence of vasopressin, we used paired-samples Student's t-test and loxitane.
CONSENT FOR TREATMENT: The undersigned hereby authorizes Doctor to take X-Rays, study models, photographs, or any other diagnostic aids deemed appropriate by Doctor to make a thorough diagnosis of the patient's dental needs. I also authorize Doctor to perform any and all forms of treatment, medication and therapy, that may be indicated. I also understand that the use of anesthetic agents embodies a certain risk. The above information provided is an accurate representation of my health and all prescription medicines are listed. I understand that dental treatment in and of it self embodies a certain risk and that additional treatment other than what initially prescribed may be required to help save my teeth. PATIENT Signature Parent of Child ; Date: DENTIST Signature.

Isation has been observed in electronics and automobiles. Other industries that have done well are fertiliser, paper and paper board, tractors, industrial chemicals, and steel.3 In order to monitor progress on poverty reduction towards the achievement of the Millennium Development Goals MDGs ; , the Planning Commission of Pakistan established its poverty line in 2000-01. This poverty line was calculated on the basis of 2, 350 calories per adult equivalent per day at Rs. 748.57 per capita per month at the prices of 2000-01 and minimum non-food requirements. Table 4 shows that the incidence of poverty fell during 1987-88 and 1990-91 from 29.2 percent to 26.1 percent, and then rose continuously in the 1990s until it reached 32.1 percent in 2001-02. This increase in poverty was the result of the severe exogenous shocks such as economic sanctions by the United States US ; following nuclear explosions by Pakistan in 1998 and drought. In this decade, Pakistan's GDP grew at about 4 percent compared to 6.5 percent in the 1980s. Inequality measured by the Gini Coefficient ; did not change but there was a rise in the absolute number of the poor. In 2005, the poverty line was raised to Rs. 878.64 per adult equivalent per month to incorporate the relatively high inflation rate 9.3 percent in 2004-05 ; . Table 5 shows that the percentage of the population living below the poverty line has fallen from 34.46 percent in 2001 to 23.9 percent in 2004-05. During 2000-05, urban poverty has declined from 22.69 percent to 14.9 percent whereas rural poverty fell from 39.26 to 28.1 percent. The substantial reduction in poverty in 2005 indicates that the impressive growth performance of Pakistan during the last four years is the major contributing factor towards poverty reduction. Strong economic growth generated numerous employment opportunities. According to Labour Force Survey 2005, since 2003-04 to the first half of 2005-06, 5.82 million new jobs have been created as against an av and loxapine. 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If do not receive the ketotifen order within 29 days about 1 % of orders will be lost at the post office ; , we shall send a new order free of charge and lyrica.
Fumarate, and VUF 8328 S-[2- 4-imidazolyl ; propyl]isothiourea dihydrobromide ; were synthesized at the Department of Medicinal Chemistry, Vrije Universiteit Amsterdam. Famotidine, ketotifen fumarate and 8R-lisuride were purchased from ICN Biomedicals Inc. USA ; , amoxapine, d-chlorpheniramine maleate, clozapine, cimetidine, N-desmethyl clozapine, diphenhydramine hydrochloride, doxepin hydrochloride, forskolin, histamine dihydrochloride, imipramine hydrochloride, loxapine, mepyramine pyrilamine maleate ; , R ; --methylhistamine dihydrochloride, S ; --methylhistamine dihydrochloride, Nmethylhistamine dihydrochloride, N-oxide clozapine, octoclothepin, pertussis toxin, polyethyleneimine PEI ; , ranitidine hydrochloride, S + ; - and R - ; -sopromidine Institute of Pharmacy, Free University Berlin ; , tripelennamine hydrochloride, and triprolidine hydrochloride were purchased from Sigma RBI USA ; . 2-Nitrophenol--D-pyranoside ONPG ; and G418 were from Duchefa The Netherlands ; , promethazine was from VUMC Pharmacy Amsterdam, fexofenadine from Ultrafine Chemicals UK ; , tiotidine.

Table 6 MULTIPLE VS. SINGLE SERVING SNACK PACK COMPARISON Based Upon 1000 Lbs. of Product ; Product Package Pdct Pkg Package Size Net Discards Lb. ; 12.9 269.1 and pregabalin and ketotifen. If the problem does not get better, your health professional may try another treatment or do more tests. However, using too much ketotifen can cause an overdose, thus doses should not be taken too closely together or at the same time and labetalol.
61 Culshaw M 2001 ; . Presentation at Managing Medicines thru' Pharmaceutical Care conference, 8 October, Birmingham.
To two-enzyme kinetics, whereas activation by Triton X-100 led to one-enzyme kinetics. N-Glucuronidation of tertiary amines is a biotransformation pathway largely restricted to humans and nonhuman primates. In a recent review, Hawes 1998 ; listed 23 antihistamines, tricyclic antidepressants, neuroleptics, and related drugs for which quaternary ammonium glucuronides were measured in human urine. The drugs with the highest percentages of the dose represented by N-glucuronides were ketotifen 24% ; and doxepin 23% ; . The enzymatic basis of quaternary ammonium glucuronide formation has been elucidated by the use of UDP-glucuronosyl transferases UGTs ; 1 expressed in cell lines. Of the more than 30 UGTs studied, only human UGT1A3 and 1A4 proved able to catalyze the N-glucuronidation of aliphatic tertiary amines review by Green and Tephly, 1998 ; . Although the substrate spectrum was similar with the two enzymes, homogenates of cells expressing UGT1A4 usually achieved higher conjugation rates Green et al., 1995, 1998; Green and Tephly, 1996 this also applied to ketotifen glucuronidation. Kinetic studies resulted in apparent KM values around or above 100 M with amitriptyline, chlorpromazine, and clozapine for UGT1A4 Green et al., 1995, 1998; Green and Tephly, 1996 ; , whereas a 1.8-fold higher value was obtained with amitriptyline for UGT1A3 Green et al., 1998 ; . However, the conju1 Abbreviations used are: UGT, UDP-glucuronosyl transferase; R ; - or S ; GlucA or B, glucuronides of R ; - or -ketotifen; ESI, electrospray ionization; CID, collision-induced dissociation; ax, axial; eq, equatorial, HH COSY, Hartman-Hahn correlation spectroscopy; HH ROESY, Hartman-Hahn rotating-frame Overhauser effect spectroscopy.
Note: the t3, ketotifen, and albuterol that i have are all from cemproducts and are research liquids.

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The Managed Prescription Drug Prescription ; Program applies to all members covered under Option A, the Out of Area Option, and the PPO Option. The Prescription Program has a Retail pharmacy component administered by Medco Health Solutions' PAID Prescriptions, L.L.C. ; and a Mail-Order component administered by Medco Health Solutions' Rx Services ; . Both are administered by Medco. All coverage under the Prescription Program is subject to medical necessity determination and other Plan limitations.
Drugs. They augment rather than reduce the stores of intrinsic norepinephrine in the myocardium.11 By contrast, with 1 exception, the electrocardiographic abnormalities were not increased in the uremic patients by the adrenolytic drugs. Rather, a partial or complete return of the electrocardiogram to normal occurred during their action, despite cardiac acceleration. This improvement suggests that excess circulating catecholamines were temporarily prevented from contributing to the sum total of injurious metabolic effects of catecholamine action otI the heart muscle. The nature of these metabolic effects of the catecholamines on the myocardium has recently been reviewed by one of us.22 In some animal experiments, 23 Dibenamine and other adrenolytic drugs diminished or suppressed the S-T and T-wave changes that were induced by infusion of catecholamines. An analogous effect occurred with combinations of norepinephrine and Regitine.24 and lamictal. Dear Governor Musgrove: On May 8, 2002, we notified you of our intent to investigate the Oakley Training School in Raymond, Mississippi "Oakley" ; and the Columbia Training School in Columbia, Mississippi "Columbia" ; pursuant to the Civil Rights of Institutionalized Persons Act, 42 U.S.C. 1997 "CRIPA" ; , and the Violent Crime Control and Law Enforcement Act of 1994, 42 U.S.C. 14141. Consistent with the statutory requirements of CRIPA, we are writing to report the findings of our investigation. At the outset, we wish to express our appreciation for the complete cooperation of the staff at the facilities, the Department of Human Services, and the Attorney General's Office during this investigation. On June 24-28, 2002 and July 16-17, 2002, we conducted onsite inspections of Oakley with expert consultants in juvenile justice administration, psychology, medicine, education, and sanitation. On July 29-August 1, 2002 and September 25-27, 2002, we inspected Columbia with all but the sanitation expert consultant. We reviewed documents including, but not limited to, policies and procedures, incident reports, medical and education files, and facility maintenance records. We find that conditions at Oakley and Columbia violate the constitutional and statutory rights of juveniles. Youth confined at Oakley and Columbia suffer harm or the risk of harm from deficiencies in the facilities' provision of mental health and medical care, protection of juveniles from harm, and juvenile justice management. There are also sanitation deficiencies at Oakley. In addition, both facilities fail to provide required general education services as well as education to eligible youth as required by the Individuals with Disabilities Education Act "IDEA" ; , 20 U.S.C. 1401, et seq., and Section 504 of the. ~~ The University of Colorado sheds some light on the relationship between the hippocampus and the neocortex in learning. Both areas are involved in learning new tasks. The neocortex processes slower, overlaps categories and attempts to find the patterns and structure of the material. The hippocampus processes faster, uses completely separate representations to code facts and details of specific events and is less prone to interference. In other words, the hippocampus memorizes, the neocortex learns. O'Reilly, R. & Rudy, J. 2000. Hippocampus, Vol 10 4 ; , 389397. ~~ A study out of Duke University shows that, if at all possible, our brain will solve tasks by processing information in only one hemisphere. We see this on simple problem solving tasks. However, as the tasks get more complex, the brain will always choose to process by coordinating information between the two hemispheres. I suppose if we want to give our brains a good wellbalanced workout, we need to do complex tasks once in a while. Weissman and Banich 2000 ; . Neuropsychology, vol 14 1 ; , 4159. ~~ Planning involves maintaining one main goal while working on subgoals for that main goal. This is apparently one of the unique human brain functions. The National Institutes of Health in Bethesda, Maryland has published findings which show that that particular task is performed in the most anterior part of the frontal lobes called the fronto polar prefrontal cortex. Koechlin et. al, Nature 1999, vol 399 6732 ; 148151. ~~ MRI, PET SPECT scans all support major mental disabilities result from adnormalities in the frontal lobes. Major depression and schizophrenia are associated with problems in the left frontal lobe. Impulsiveness and mania in the right frontal lobe. Obsessivecompulsive behaviors tend to result from problems in the orbital frontal lobes. Joseph. Psychiatry: Interpersonal & Biological Processes, 1999, v 62, 2, 138172. DEPRESSION ~~ A substantial number of adolescents with epilepsy and seizures have clinical depression as well, so says the Journal of Epilepsy & Behavior. The prevalence of depression among these teens has not been widely known and many are not being treated. Researcher Benjamin Hankin writes on how the role of stressors in a young person's life interact with genetic, biological, cognitive and interpersonal factors to predict depression. Hankin, B. 2006 ; . Epilepsy & Behavior, Vol .8 1 ; , 102114.
10, 1996, Barron's published an article titled Hooked on Drugs: Why Do Insurers Pay Such Outrageous Prices for Pharmaceuticals? describing AWP as "Ain't What's Paid." See DX 2641. ; It stated. Methods: fifty asthmatic children were enrolled and received 8 weeks of treatment with oral montelukast sodium 5mg chewable tablet administered once daily, or 1mg ketotifen, and were followed for a 4-week post-treatment washout period.

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Although the majority of studies overwhelmingly point toward the safety of HGH, there is, as in virtually any area of medical science, some conflicting data among studies that confuses the issue.This is true because of the complex nature of the human body and its physiology -and the truism that medicine is not an exact science. As in all aspects of medical therapeutics, each of us must evaluate the information that is available, along with our needs and desires and measure these against the potential risk, if any. Your Anti Aging Group doctor and counselor can help you understand and evaluate all the information available with as little prejudice as possible. Obviously we feel that for most patients the benefit of HGH therapy far outweighs the risk- otherwise we would not be pursuing this type of medical practice. Only you however, can make the decision for yourself. To put it into context, similar controversy has surrounded the use of estrogen in post-menopausal women for the past 30 years.We now know that estrogen replacement in women may increase the risk of breast and endometrial cancer in some women with family histories of breast cancer, and other risk factors. However, because of estrogen's proven protective effects against many other diseases alzheimer's disease, heart disease, osteoporosis and colon cancer ; , overall mortality is lower in women who take estrogen than in those who do not. It will be many years before we have as much data on HGH as we do estrogen, but we feel that for most people who have low IGF-1 levels, the benefits of HGH replacement therapy outweigh the risks.
We started to hold patient focus groups in September 2003 in association with the Day Surgery Unit. Following recommendations from our Charter Mark assessment we decided to make the arrangements for these groups more focused. We decided that it would be beneficial to hold two or three per year. These groups would be for ex-patients or relatives who had been on the Intensive Care Unit. The purpose of the focus groups would be to elicit the experiences of the patient or carer while they were on the unit and since discharge. Any comments, thoughts or suggestions would be fed back to the quality group. In this way we could action any suggestions. Each group lasts for an hour and is focused around the questions outlined in Table 1. The first group consisted of five ex-patients and a carer. They were all eager to talk about their experience. They also had lots of unanswered questions. All of the ex-patients had been on the unit for longer than two weeks. They were all ventilated and had been given some form of sedation. They acknowledged that their memories were very disorientated and confusing. At times it was hard for them to distinguish between reality and fiction. We did however try to capture some of their memories and experiences: Dreams All of the ex-patients experienced some form of dreams. They were unsure if the dreams happened before or after consciousness. Some of the dreams were pleasant; others had dark dreams. Some of the ICU staff featured in the dreams. In trying to explain this they felt that perhaps they surfaced into reality and registered a staff member's face so when they slipped into a dream-like state the staff member's image featured in the dream. A patient whose bed was opposite the staff noticeboard felt that the faces were watching him and appeared to come alive. They mentioned that seeing photos of their family was very comforting. Information Some of the patients were unsure if they were told what happened to them. Others found that it was very helpful for staff to explain what they were doing. The carer felt especially comforted by the staff talking to their relative. STAMIS UPDATE Medical Maintenance Soldiers manage medical equipment utilizing STAMIS systems, ULLS-G [legacy system], SAMS-1, and SAMS-2. SAMS-E combination of ULLS-G and SAMS-1 ; and SAMS-2 will be the next generation maintenance management system, and is currently being fielded by CASCOM. The medical maintenance community is currently using an Equipment Master File EMF ; , which is a modified Master Maintenance Data File MMDF ; . As of Jan 06, there were 66 end items on the MMDF. On 5 May 06, an unscheduled publication of the MMDF was released by LOGSA, we currently have 117 items in the MMDF. Through USAMMA, we added approximately 450 plus maintenance significant end item into the SAMS-2 box. Thus, the EMF was produced for the STAMIS users to load into their system. The issue, the work order register data could not be roll up to LOGSA, because the end items are not cataloged at LOGSA, therefore no global visibility of our assets and no data history. Each maintenance manager, unit commander, higher level support, materiel developer, and LOGSA could not query past or present history data from a centrally located data warehouse, which could play a vital role of the life cycle management of today's modern medicine and support strategy. That's why we have created an alternative solution to gain the visibility of all our assets in CONUS, OCONUS, and downrange theater. In order for our STAMIS systems to operate efficiently, we need the following elements: an equipment catalog or baseline, the MMDF an unit address for each STAMIS system Derivative Unit Identification Code DUIC means to transmit data or system configuration FTP, IP address property book or list of equipment to support mission; and a STAMIS operator. The Master Maintenance Data File MMDF ; consists of the basic cataloged data elements of each maintenance significant medical end item, which is comprised of the following items: Long and Short Nomenclature, FSC, NIIN, ECC, EIC, MATCAT, AAC, LIN, Model #, Manufacturer, EICC, Commodity type, Reportable, NMC. - The key data fields necessary to get our equipment cataloged into the MMDF is the LIN and the EIC. Since many of our end items do not have a LIN assigned, LOGSA has agreed to provide our medical equipment with pseudo LIN and EIC or O-LINs and EICs. NOTE: ULLS-G can only accept a particular LIN format, one alpha character and 5 digits ; , i.e. A12345. NOTE: SAMS-E will accept the regular LIN format and the Non-Standard LIN NSLIN ; format two alpha characters, 3 digits, and an alpha or digit as the last character ; , i.e. ZA0583, ZA054E, FJ259Y, ZA054X, ZA0541, see explanation below. Why O-LINS exist in MMDF. The primary reason is that the ULLS-G legacy system cannot process records with standard or non-standard LINs in any format other than one alpha character followed by five numeric characters. If an item that needs to be added to MMDF has a non-standard LIN other than the aforementioned format, a LIN that is usable by the ULLS-G legacy system must be assigned so the added records will not reject. This is why O-LINs are created. They are assigned and approved by MMDF and other personnel at LOGSA. When it is necessary to assign an O-LIN, an effort is made to make it look as similar to the non-standard LIN as possible. Thus far there have been three LIN formats for which O-LINs had to be assigned. Please see the examples below: * Two alpha characters followed by four numeric characters. Example: AB1234.