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Introduction: Persistent Hyperplastic Primary Vitreous PHPV ; can have wide clinical presentations. Most cases tend to be unilateral though review of various cases from the literature shows it to be present in both eyes of 11% of cases. 1. The cases that present bilaterally have associated systemic anomaly and die at a younger age. Our patient has no systemic abnormality; a similar case of bilateral PHPV without any systemic disease has been reported earlier. 2. Buphthalmos due to secondary glaucoma is an important late presentation of PHPV and has been reported in 26% of the cases. 3. One eye of our patient had buphthalmos with IOP of 19.4mmHg. Glaucoma in PHPV could result from recurrent vitreous hemorrhage, which is the most likely mechanism in our case 3. Aim of the study : To present as poster a rare case of bilateral PHPV with buphthalmos in one eye. Method: A five-month-old male infant was seen in out patient with history of bilateral leucocoria since the age of four months. Results: Examination of eyes under general anesthesia showed: Right eye corneal diameter of 14mm in horizontal and 13.5mm in vertical meridian. Anterior chamber was shallow. Iris showed blood vessels going from iris to the lens surface with ectropion uvea. The lens was clear. There was no view of the fundus other than a red glow from dense vitreous hemorrhage. Intraocular pressure was 19.4mmHg with Schioz tonometer. Left eye: Corneal diameter was 9.5 mm in horizontal and vertical meridian. Anterior chamber was normal. Lens was clear. There was a fibrovascular membrane on the posterior surface of the lens obscuring the fundus view. Intraocular pressure was 14.6mmHg. B scan ultrasonography of the right eye was suggestive of vitreous hemorrhage with a fibrous band from disc to posterior lens surface. Left eye scan showed a similar hyper echoic shadow from disc to posterior lens surface suggestive of fibrous band from disc to posterior lens surface. MRI scan on T1 image showed hyper intense echoes from vitreous cavity of right eye and central hypo intense echoes from disc area to the posterior lens surface in both eyes . Histopathology of the right eye showed dense fibro vascular band extending from disc to posterior surface of lens confirming diagnosis of PHPV. The left eye underwent pars plana lensectomy with vitrectomy. After lensectomy ciliary processes were seen to be dragged towards the center and retina was thrown into fixed folds and was incarcerated into the fibro vascular band. Conclusion: Bilateral PHPV is a rare disorder and sometimes can present as secondary glaucoma due to recurrent vitreous haemorrhage. References : 1. Haddad R, Font R, Reeser F. Persistent hyperplastic primary vitreous: a clinicopathological study of 62 cases and review of literature. Surv ophthal 1978; 23: 123-134. Magnus H. Doppelseitiges Pseudogliom, yorgetauscht durch Bindegewebsbildung hinter der Linse mit Arteria hyaloidea persistens bei Mikroophthalmos. Graefes Arch Exp Ophthamol 1927; 59: 271-286. Alward WLM, Krasnow MA, Keech RV, Pulido JS et al Persistent hyperplastic primary vitreous with glaucoma presenting in infancy. Arch Ophthalmol 1991; 109: 1063-1064. Order on file; submit claim only CMN 04.01 submitted with initial claim Order on file with any other appropriate documentation to justify use. Substantiate medical necessity. ; Submit claim only and mesterolone.
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ACADEMIC TRAINING 1967-1971 St. Joseph's University, Philadelphia, Pennsylvania. B.S. in Chemistry. 1971-1975 Texas A&M University - College Station, Texas. Received Ph.D. in Bio-Organic Chemistry see below for minor ; . 1972-1974 Baylor College of Medicine - Houston, Texas. Department of Microbiology. Graduate Minor in Medical Microbiology and Biochemistry. EXPERIENCE TEACHING 9 71 - 6 Texas A&M University - Graduate Teaching Assistant, Dept. Chemistry. 10 77 - 6 The Ohio State University - Assistant Professor, Depts. Radiology and Pharmacology. 7 82 - 6 The Ohio State University - Associate Professor, Depts. Radiology and Pharmacology. 7 85 - present The Ohio State University - Professor, Depts. Radiology and Pharmacology. RESEARCH 6 72 - 6 present and phentermine.

James Salzer MD, PhD New York University Medical Center New York, NY NMSS Area: New York City Chapter Award: Research Grant Term Amount: 4 1 06-3 7, 079 2005 Stephen C. Reingold Research Award "Regulation of CNS myelination and remyelination by neuregulin1" Determining the role of a molecule in myelin formation and repair, for clues to developing repair strategies in people with MS. In multiple sclerosis, the myelin sheaths that insulate nerve fibers are broken down by immune cells, resulting in damage and loss of nerve fibers also known as axons ; , abnormalities of nerve impulse conduction and significant neurologic disability. For reasons that are not well understood, once myelin is damaged, it is poorly repaired. Researchers have demonstrated that the survival of the myelin sheath is actually regulated by signals from the axon. James L. Salzer, MD, PhD, is studying the mechanisms by which axons regulate myelin formation in order to learn more about molecules that may be affected by MS, and to develop strategies for repairing myelin in this disease. Dr. Salzer's team has determined that a "growth factor" found on the surface of axons neuregulin1 strongly promotes myelin formation. Now they are studying whether neuregulin1 is required to initiate myelin formation and maintenance, by examining mice in which neuregulin can be deleted at various stages. The team is also inducing myelin damage in these mice and tracking subsequent repair processes to determine whether neuregulin plays a role in myelin repair.

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