Young, S. D.; Britcher, S. F.; Tran, L. O.; Payne, L. S.; Lumma, W. C.; Lyle, T. A.; Huff, J. R.; Anderson, P. S.; Olsen, D. B.; Carroll, S. S. Antimicrob. Agents Chemother. 1995, 39, 2602. Klebe, G.; Abraham, U.; Mietzner, T. J. Med. Chem. 1994, 37, 4130. Bohm, M.; Sturzebecher, J.; Klebe, G.; J. Med. Chem. 1999, 42, 458. Pungpo, P.; Hannongbua, S. J. Mol. Graphics & Modell. 2000, 18, 581. Ravichandran, V.; Agrawal, R. K. Bioorg. Med. Chem. Lett. 2007, 17, 2197. Barreca, M. L.; Carotti, A.; Chimirri, A.; Monforte, A. M. Bioorg. Med. Chem. 1999, 7, 2283. Nair, A. C.; Jayatilleke, P.; Wang, X.; Miertus, S.; Welsh, W. J. J. Med. Chem. 2002, 45, 973. Jayatilleke, P. R. N.; Nair, A. C.; Zauhar, R.; Welsh, W. J. J. Med. Chem. 2000, 43, 4446. Hilgeroth, A.; Fleischer, R.; Wiese, M.; Heinemann, F. W. J. Computer-Aided Mol. Design 1999, 13, 233. A. K. Debnath, J. Med. Chem. 1999, 42, 249. Oprea, T. I.; Waller, C. L.; Marshall, G. R. Drug Design Discovery 1994, 12, 29. Buolamwini, J. K.; Assefa, H. J. Med. Chem. 2002, 45, 841. Raghavan, K.; Buolamwini, J. K.; Fesen, M. R.; Pommier, Y.; Kohn, K. W. J. Med. Chem. 1995, 38, 890. Sahu, K. K.; Ravichandran, V.; Mourya, V. K.; Agrawal, R. K. Med. Chem. Res. 2007 In press ; . Debnath, A. K.; Jiang, S.; Strick, N.; Lin, K.; Haberfield, P. J. Med. Chem. 1994, 37, 1099. Arranz, M. E.; Diaz, J. A.; Ingate, S. T.; Witvrouw, M.; Pannecouque, C.; Balzarini, J.; Clercq, E. D.; Vega, S. Bioorg. Med. Chem. 1999, 7, 2811.
We don't know how many of the 26 subjects in this study who were tested at 6 months received at least 9 shocks at 2.5 times threshold. Less than 20? Less than 10? This study cannot be considered to even approach being conclusive. Indeed, three years after his 1991 study Coffey himself wrote, "Clearly, prospective studies that compare pre- and post-ECT imaging data are required to determine whether ECT causes changes in brain structure."153 And were those "required" studies ever done? No. A Medline search under "Electroconvulsive and MRI" failed to uncover a single post-Coffey 1991 ; prospective MRI study of ECT's effects on brain structure. Nevertheless, other ECT researchers are happy to write about this issue as if it has been completely resolved. In July, 1994, we find Devanand et al. claiming, "There is no credible evidence that ECT causes structural brain damage, "154 even though, 1 ; He admits "there is a paucity of quantitative data from well-designed [CT and MRI] studies using sensitive methods."155 2 ; Coffey is, at that very same time mid-1994 ; , saying that prospective studies are needed and, 3 ; There was credible evidence in a ; Coffey et al., 1988, in which he states that one explanation of his evidence was that ".ECT may have caused cerebral atrophy."156 b ; Coffey et al., 1991, reviewed above. c ; Andreasen et al., 1990, which found a correlation between the number of previous ECT treatments and increased lateral ventricular volumes loss of brain tissue ; measured by MRI.157 d ; Dolan, RJ., et al., 1986, a CT scan study which found, "Patients with a past history of treatment by electroconvulsive therapy showed more sulcal widening in the parietal and occipital areas than those not so treated."158 The authors explained that, "There was no evidence of association between the presence of these changes [sulcal widening in frontal, temporal, and interhemispheric areas of the brain] and a family history of depression, the duration of depressive illness, the age of onset of illness, the course of illness or exposure to psychotropic medication."159 There was also no association with age or alcohol use. Coffey et al. 1991, in reviewing previous brain scan research, left this study out but did include a 1985 Dolan study which found no association between ventricular size and ECT. e ; Calloway et al, 1981, which reported that frontal lobe atrophy was significantly more common in elderly depressed patients who had received ECT than in those who had not.160 f ; Weinberger et al., 1979, which found that cortical atrophy was significantly more common in schizophrenic patients who had received ECT than in those who did not.161 34.
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From the 1Section of Cardiology, Department of Medicine, Karolinska Hospital, Stockholm, Sweden; and the 2Section of Clinical Physiology, Department of Surgical Sciences, Karolinska Hospital, Stockholm, Sweden. Address correspondence and reprint requests to Professor Helene von Bibra, Section of Cardiology N3: 05, Karolinska Hospital, SE-17176 Stockholm, Sweden. E-mail: helene.vonbibra ks . Received for publication 21 February 2002 and accepted in revised form 12 July 2002. CV, coefficient of variation; MBFI, myocardial blood flow index; PET, positron emission tomography; SI, signal intensity; SImax, myocardial blood volume; Va, late diastolic velocity; Vd, velocity during early diastole; Vs, velocity during systole. DIABETES, VOL. 51, OCTOBER 2002.|
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PREVENTING INFLUENZA Vaccination is the primary method of preventing influenza infection or lessening the severity of influenza illness. Because circulating influenza strains change from year to year, a new vaccine is manufactured each year. To optimize protection, individuals must receive a new influenza vaccine annually. October to November is the ideal time for influenza vaccination, especially for persons at higher risk of developing complications from influenza, such as residents of long-term care facilities LTCF ; . The Advisory Committee on Immunization Practices ACIP ; recommends that vaccination be routinely provided to residents with the concurrence of attending physicians. Consent should be obtained from the resident or family member at the time of admission or anytime afterwards. In addition, all residents should be vaccinated at one time before the start of flu season and those admitted through March after the completion of the facility's vaccination program should be vaccinated at the time of admission. A rule from the Centers for Medicare and Medicaid Services CMS ; recently removed the physician signature requirement for the administration of influenza and pneumococcal vaccines to Medicare and Medicaid patients in hospitals, long-term care facilities, and home health agencies.1 However, to comply with Georgia Pharmacy law O.C.G.A. 26-4-5, 26-4-80 ; , both vaccines must be issued in the same manner as all "legend drugs". Provisions for these vaccines should be governed by the policies and procedures of the facility per administrative direction on legend drugs. Health-care workers at LTCF are also strongly urged to be vaccinated to reduce the chance of their spreading influenza to residents. In fact, given that influenza vaccine is more effective at preventing disease among younger, healthy adults than among the elderly, vaccination of workers may be more effective in preventing disease among LTCF residents than vaccination of the residents themselves. A limited supply of a live, attenuated intranasal influenza vaccine recently approved by the Food and Drug Administration FDA ; will also be available for the 2004-2005 season. This intranasal vaccine is licensed for use only in healthy children and adults ages 5-49 years, and will contain the same influenza strains as the inactivated vaccine. Listed below are the manufacturers of influenza vaccines with contact information for ordering. The ideal time to order influenza vaccine for the upcoming flu season is in the early spring, as most manufacturers' stocks are pre-booked by May. Inactivated injectable influenza vaccine manufacturer: Aventis Pasteur 1-800-822-2463 Live, attenuated intranasal influenza vaccine manufacturer: MedImmune, Inc. 1-301-398-0000 and triphasil.
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Twelve C57 BL6J mice Charles River Laboratories, Inc, Wilmington, Mass ; aged 6 to 8 weeks were anesthetized with methoxyflurane inhalation 0.5 mL titrated ; and shaved, and their skin was prepared with povidone-iodine Betadine; Purdue Pharma LP, Stamford, Conn ; . Eight-millimeter wounds were created bilaterally on the dorsum of each animal with the use of an 8-mm dermal punch biopsy forceps Miltex Instrument Co Inc, Bethpage, NY ; , leaving the panniculous carnosus muscle intact. A transparent, sterile dressing was then applied circumferentially around the trunk of the animal Tegaderm; 3M Health Care, St Paul, Minn ; . Wounds were allowed to granulate for 3 days before intervention. The animals were then divided into 4 groups, and wounds were treated with a preparation of either 0.3% ciprofloxacin with 0.1% dexamethasone CiproDex; Alcon Laboratories, Inc, Fort Worth, Tex [trade name licensed to Alcon Laboratories, Inc, from Bayer Ag, Leverkusen, Germany] ; , 0.3% tobramycin with 0.1% dexamethasone TobraDex; Alcon Laboratories, Inc ; , 0.2% ciprofloxacin hydrochloride with 1% hydrocortisone Cipro HC; Alcon Laboratories, Inc ; , or phosphate-buffered saline PBS ; n 6 wounds per group ; . One hundred microliters of each preparation was applied once daily for 3 consecutive days by injection into the wound bed with the use of a 1-mL syringe and a 30.5-gauge needle. A transparent, sterile dressing was placed over the wound to prevent the preparation from "rolling off" of the granulation tissue bed. Animals were euthanized by carbon dioxide inhalation followed by cervical dislocation, and all wounds were harvested on day 7 in preparation for tissue sectioning and ultram.
Tetracycline caps . 7 TEXACORT soln 2.5% . 28 THALITONE 15 mg . 24 THALOMID . 37 THEO-24. 43 theophylline . 43 theophylline ext-rel tabs . 43 THERACYS . 14 THIOGUANINE . 14 thioridazine . 17 thiotepa. 13 THIOTEPA 30 mg . 13 thiothixene. 16 THORAZINE supp . 10, 17 TIAZAC 420 mg . 23 TIKOSYN . 22 TILADE . 43 timolol maleate. 39 timolol maleate gel. 39 TINDAMAX . 16 tizanidine. 43 TOBI . 43 TOBRADEX. 38, 39 tobramycin . 38 TOBREX oint. 39 TOPAMAX .8, 13 TOPROL-XL . 19, 23 torsemide . 24 TRACLEER. 26, 43 tramadol. 6 tramadol acetaminophen . 6 TRANSDERM SCOP . 10 tranylcypromine. 9 TRAVATAN . 39 trazodone . 10 TRELSTAR. 35 tretinoin . 29 triamcinolone acetonide crm, lotion, oint 0.025% . 28, 33 triamcinolone acetonide crm, lotion, oint 0.1% . 28, 33 triamcinolone acetonide crm, oint 0.5% . 28, 33 triamcinolone paste . 26 triamterene hydrochlorothiazide . 24 TRICOR . 24 trifluoperazine . 17 trifluridine. 39 trihexyphenidyl. 16.
Major source of omega-3 for Americans is plants; soybean oil is a major omega-3 source. with better weight loss than a 20% fat diet; the percent of fat in the diet may not be that important. A very low fat etable fat lowered breast cancer risk. There are a lot of hormones in milk, even natural diet increased breast cancer risk; olive oil & increased vegones. Cows have been bred so that they have higher milk and valtrex.
While such large studies depict ADHD as a common comorbidity among adults with bipolar disorder along with other psychiatric conditions ; , the rate at which these two illnesses are diagnosed together in the real world of clinical practice does not reflect the research data.4 In an analysis of 2005 medical and prescription databases in the US, adult patients aged 18 and older who had a new ICD-9 diagnosis for ADHD, depression, or bipolar disorder were tracked for claims citing one or more of the other targeted conditions during the study period. Data were then compared with the prevalence data generated by the NCS-R.1 In patients with a primary diagnosis of bipolar disorder, only 2.5% received a comorbid diagnosis of ADHD, and only 1.7% received a comorbid diagnosis of both depression and an anxiety disorder. In the NCS-R, ADHD was most frequently comorbid in those with a primary diagnosis of depression 32.0% ; , followed by bipolar disorder 21.2% ; and an anxiety disorder 9.5% ; Figure 1 ; .1 The significantly lower co-diagnosis rates, based on current claims data, imply that adult ADHD is being identified as a comorbidity of bipolar disorder far less often than studies suggest should be the case. Adult patients presenting with primary psychiatric diagnoses should routinely be screened for comorbid ADHD. DIAGNOSING ADHD IN BIPOLAR ADULTS Diagnosing ADHD in adults presents challenges because hyperactive symptoms are typically less prominent in adults than in children. One study, for example, measured ADHD symptoms in 128 boys five times over a 4-year period.11 While increasing age was significantly associated with decline in total ADHD symptomatology, symptoms of inattention continued into adulthood more frequently than did symptoms of hyperactivity or impulsivity. In the oldest age group 18 to 20 years ; , the prevalence of syndromal ADHD remission--that is, loss of full DSM-IV diagnostic status--was greater than 60%. However, the rate of functional ADHD remission was only 10%--that is, while the patient no longer qualified for full ADHD diagnostic status, symptoms of inattentiveness persisted past age 20 and well into adulthood. Diagnosing ADHD in bipolar adults presents even more formidable challenges. ADHD assessment is best conducted when a bipolar patient is euthymic.10 Otherwise, the symptoms associated with mood changes could be confused with.
Atsushi Minami, Shinji Hirose, Tomohiro Nomoto, Shoichiro Hayakawa, Internal Medicine, Kawasaki Kyodo Hospital, 2-1-5 Sakuramoto Kawasaki City 210-0833, Japan Correspondence to: Atsushi Minami, Kawasaki Kyodo Hospital 2-1-5 Sakuramoto Kawasaki City 210-0833, Japan minamiad1dionnep -281 -281 Telephone: + 81--281 Fax: + 81--281 Received: 200-01-2 Accepted: 200-02-1 lithotripsy. World J Gastroenterol 2007; 13 15 ; : 2179-2182 and vasotec.
Seek help from trained health worker if you notice rapid weight loss or if the sick person consistently refuses to eat any food or is not able to swallow.
TABLE 4. tice ; * Knowledge of EC n 233 Pediatricians in Pracn Knowledge Unable to identify any FDA-approved regimen Knows maximum time of prescribing General physical examination Necessary before prescribing Not necessary but advisable Not necessary Pelvic examination Necessary before prescribing Not necessary but advisable Not necessary Pregnancy test Necessary before prescribing Not necessary but advisable Not necessary Informed consent by parent or guardian Necessary before prescribing Not necessary but advisable Not necessary * Not all respondents replied to each question. 164 62 103 % 72.9 ; 27.9 ; 46.8 ; 36.4 ; 16.8 ; 35.7 ; 37.1 ; 27.1 ; 63.8 ; 16.7 ; 19.5 ; 10.0 ; 25.1 ; 64.8 and verapamil and tobradex.
Drug Name TEMODAR PA ; TEMOVATE temozolomide TENEX tenofovir TENORETIC TENORMIN TEQUIN TERAZOL terazosin terbinafine HCL terbutaline terconazole vaginal TESLAC TESSALON TESTODERM testolactone testosterone tetracycline TEVETEN TEVETEN HCT THEO-24 THEOLAIR theophylline theophylline CR theophylline SR thiabendazole THIOGUANINE THIOLA thioridazine thiothixene tiotropium THORAZINE thyroid THYROLAR tiagabine TIAZAC TICLID ticlopidine TIGAN PDL Section 2-A 5-H 2-A Drug Name TIKOSYN TILADE timolol maleate timolol maleate ophth timolol ophth TIMOPTIC TIMOPTIC XE tipranavir tiopronin tiotropium tizanidine TOBRADEX tobramycin ophth tobramycin-dexamethasone ophth TOBREX tocainide TOFRANIL TOFRANIL tolazamide TOLBUTAMIDE TOLECTIN TOLINASE tolmetin sodium tolterodine SR tolterodine. TONOCARD TOPAMAX TOPICORT topiramate TOPROL XL TORADOL toremifene citrate torsemide TRACLEER tramadol tramadol-APAP TRANDATE trandolapril trandolapril-verapamil TRANSDERM-SCOP PDL Section 3-E 12-D 3-C.
New "stable liquid" technology could provide significant savings in the costs of immunisation programmes by removing the need for vaccines to be refrigerated. Cambridge Biostability Ltd has developed a technique whereby vaccines are embedded in sugar beads or microspheres and suspended in an inert liquid where they can be stored indefinitely without refrigeration. After injection, the beads dissolve in bodily fluids to release the vaccine. The company says that currently 50 per cent of all vaccines are wasted partly due to suspected or real temperature damage. It adds that within existing budgets savings from removing the "cold chain" alone will enable the vaccination of an extra 10 million children worldwide. CBL has received a grant of 950, 000 from the Department for International Development to develop a pentavalent vaccine diphtheria, tetanus, pertussis, haemophilus influenzae type b and hepatitis B ; using this technique, in collaboration with Panacea Biotec, based in New Delhi. This technology could also be used to develop slow release vaccines, by adapting the sugar to dissolve over time which may overcome the need for boosters and vicoprofen.
The possible ameliorative effects of CFT on FMS may involve mechanisms as diverse as improved muscle blood flow or CNS-induced hypoalgesia, felt to be related to activation of endogenous opioids. Vigorous exercise also induces increases in adrenocorticotropic hormone ACTH ; and cortisol, which may also promote analgesia. The only controlled report of EMG biofeedback training randomized 12 patients with fibromylagia to EMG biofeedback or sham biofeedback.273 The EMG biofeedback group had a significant improvement in pain, morning stiffness and tender points. Hypnotherapy was found to be better than physical therapy in 40 patients with refractory FMS. The hypnotherapy group demonstrated better outcome in pain, fatigue, sleep and global assessment, but not in tender points.274 Goldenberg has found that a mindfulness meditation-based relaxation response program is helpful in FMS. Other less wellstudied non-medical treatments include transcutaneous electrical nerve stimulation TENS ; , acupuncture, laser treatment, and tender point injections. A new modality called cranial electrotherapy has recently been proposed as a treatment for FMS.275 It has been found to improve quality of life and sleep, as well as to decrease anxiety as much as 100% in a double-blind controlled trial. Its benefit in alleviating pain, however, has been less dramatic, but beneficial. A recent article described the effectiveness of electroacupuncture in relieving symptoms of FMS, including pain threshold, pain on visual analog scale, and sleep 39.
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Arrebola J, - Frankston Rehabilitation Unit Client Centred Goals in Rehabilitation. Barnfield J, Ciotta A, Friebe L, Keeble-Devlin B, Macfarlane S. Development and use of a multi-sensory environment in a designated room in an aged acute psychiatric inpatient facility. Hellier W, - Ward 4 Mt Eliza Exploration of the issues related to admission of patients for investigation of falls. Lester B MEPACS Personal Assistance Call Service Evaluation of MEPACS falls management. McCauley K, Elsom S, Keeble-Devlin B. An investigation of the efficacy of practice development and clinical supervision for mental health nurses.
PRODUCTION Surprisingly intimate feel in a live performance from Lincoln Center. Sets seem scaled to Glyndebourne despite the large theater. Costumes and stage movement are strictly traditional. PERFORMANCES Bergeson leads a reading on the light side of conventional. Orchestra, chorus, and soloists are all responsive and careful, but no spark is struck. None of the major soloists is memorable for positive or negative contribution; overall, this is a solid but forgettable performance. Unfortunately, it appears that the cast must enjoy so witty an opera for the home audience to have fun with it. TECHNICAL COMMENTS Fine video quality for a live performance. Audio seems lacking in higher frequencies, perhaps contributing to the lack of sparkle as we listen. Camera work is fine, though it appears to have been blocked at Glyndebourne. Nothing in this performance stands out; in a work less frequently performed and recorded, that might be an asset.
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At night gain more weight than animals given an equal number of calories spread throughout the day. The body secretes more insulin for the same carbohydrate intake as the day progresses. This may cause calories consumed late in the day to be stored as fat. It's acceptable to distribute your calories equally at each meal, but you'll lose weight more efficiently if you get most of your calories at breakfast and lunch and fewer at dinner.
Fallon Community Health Plan soon will be introducing the Healthwise Knowledgebase on its Web site, fchp . With this tool, for example, you and your patients may research diagnosed conditions, medications and treatment options. The content is generated from a variety of reliable resources, including the National Cancer Institute, the National Organization of Rare Disorders and the American Self-Help Clearinghouse. The Healthwise Knowledgebase is a reliable, comprehensive resource to help people be informed about their health care. Informed patients are more likely to understand their condition and take better care of themselves, as well as develop a more interactive relationship with their doctor. Watch for more information in a future issue of Connection.
Tetanus toxoid 43 TETANUS TOXOID, ADSORBED 43 tetracycline 22 THALITONE 34 THALOMID 27, 43 theophylline sustained release capsule 46 theophylline sustained release tablet 46 THIOGUANINE 27 THIOLA 38 thioridazine 28 thiothixene 28 TIKOSYN 34 TILADE oral inhaler 46 timolol maleate ophthalmic gel forming solution .44 timolol maleate ophthalmic solution 45 tizanadine tablet only 47 TOBI nebulization solution * 22 TOBRADEX ophthalmic 45 tobramycin ophthalmic solution 45 TOBREX ophthalmic ointment 45 tolmetin 20, 25 TOPAMAX 23 TOPROL XL .34 TRACLEER 34, 46 tramadol 20 TRANSDERM SCOP 24, 37 TRAVATAN ophthalmic - 2.5ml bottle 45 trazodone 24 tretinoin topical 36 triamcinolone acetonide topical 36 triamcinolone in orabase oral paste 34 triamterene hydrochlorothiazide capsule 34 triamterene hydrochlorothiazide tablet 34 TRICOR 34 trifluoperazine 28 trifluridine ophthalmic 45 trihexyphenidyl 28 TRILEPTAL 23 trilyte NULYTELY equivalent ; 37 trimethobenzamide 300mg capsule * 24 trimethoprim 22 trinessa ORTHO TRI-CYCLEN equivalent ; 41 tri-previfem ORTHO TRI-CYCLEN equivalent ; 41 tri-sprintec ORTHO TRI-CYCLEN equivalent ; 41 trivora TRIPAHSIL equivalent ; 41 TRIZIVIR 29 TRUSOPT ophthalmic 45 TRUVADA 29 16.
Eighty-eight 98.9% ; of 89 isolates M. tuberculosis 87, MOTT 1 ; were tested for resistance to 40 g RIF and all M. tuberculosis were susceptible to this concentration. The isolate from one patient was no longer viable when sub-cultured. M. tuberculosis isolates resistant to both INH and RIF i.e. multidrug resistant strains ; were not detected during this period.
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Statement of the Problem: What Is CML Blast Crisis? Chronic myeloid leukemia CML ; in blastic phase is the transition of CML in chronic or accelerated phase to an acute leukemia, characterized by 30% blasts in the bone marrow or peripheral blood, or the development of extramedullary disease outside of the spleen. In light of recent changes in the World Health Organization definition of acute leukemia, the percentage of blasts required for CML in blastic phase may someday be reduced to 20%. Consistent with the early stem cell nature of CML, 1 blastic transformation may be myeloid, lymphoid, or undifferentiated mixed, with myeloid blast crisis being about two times more common than lymphoid. Although the definition of CML in blastic phase has not changed from the pre- to post-imatinib era, the treatment history of our current CML patient population has: an ever-increasing proportion of patients with CML will have been managed exclusively with imatinib or other ABL kinase inhibitor therapy. Such a significant change in therapeutic approach will almost certainly affect the kinetics and molecular phenotype of CML blast crisis. Pathophysiology of CML Blast Crisis-- Why Does It Occur? The pathophysiology of CML blast crisis is incompletely understood. However, recent advances in our understanding of cancer and leukemia stem cell biology and emergUniversity of Texas Southwestern Medical Center Correspondence: Robert Ilaria, Jr., MD, Medical Advisor, Lilly Research Laboratories, Lilly Corporate Center DC 2133, Indianapolis, IN 46285; Phone 317 ; 433-4759, Fax 317 ; 2769666, ilariaro lilly.
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